Modification of Arruda's Accessory Pathway Localization Method to Improve the Performance of WPW Syndrome Interventions
Sándor Miklós Szilágyi (Hungarian Science University of Transylvania, Romania), László Szilágyi (Hungarian Science University of Transylvania, Romania), Constantin T. Luca (University of Medicine and Pharmacy, Romania), Dragos Cozma (University of Medicine and Pharmacy, Romania), Gabriel Ivanica (University of Medicine and Pharmacy, Timisoara, Romania) and Zoltán Benyó (Budapest University of Technology and Economics, Hungary)
Copyright: © 2008
The Wolff-Parkinson-White (WPW) syndrome is characterized by an accessory pathway (by-pass tract) between the atria and ventricles that conducts parallel with the atrioventricular (AV) node-His bundle, but faster (Wolff, Parkinson, & White, 1930; Yee, Klein, & Guiraudon, 1995). An accessory AV connection can conduct in both directions. The presence of these bypass tracts may predispose to atria-ventricular reentrant tachycardia. Moreover, in the setting of atrial fibrillation, the WPW syndrome may cause a catastrophically rapid ventricular response with degeneration to ventricular fibrillation (VF).
Key Terms in this Chapter
Accessory Pathway: An abnormal electric conduction route that connects the atria with the ventricles.
Atria: The chambers of the heart that collect the venous blood.
QRS Complex: A structure on the ECG signal that corresponds to the depolarization of the ventricles.
Electrocardiogram (ECG): A graphic produced by an electrocardiograph, which records the electrical activity of the heart over time.
Wolff-Parkinson-White (WPW) Syndrome: A syndrome of pre-excitation of the ventricles of the heart due to an accessory pathway that causes an abnormal electrical communication from the atria to the ventricles.
Ventricles: The pumping chambers of the heart.