Yoga Therapy on Digestive Function in Inflammatory Bowel Disease

Yoga Therapy on Digestive Function in Inflammatory Bowel Disease

Soccalingam Artchoudane (Center for Yogic Sciences, Aarupadai Veedu Medical College and Hospital, Vinayaka Mission's Research Foundation, India)
DOI: 10.4018/978-1-7998-3580-6.ch006
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Abstract

Inflammatory bowel disease (IBD) is a psychosomatic disorder characterized by chronic inflammation of the gastrointestinal tract. Metabolism of an individual affected with IBD is equated to imbalance of jatharagni (digestive fire) which results in atijeernam (hyper digestive disorder), ajeernam (hypo digestive disorder), or kutajeernam (erroneous digestive disorder). Yoga stabilizes jatharagni that helps energy transformation of 1) food substances into nutritious substance, 2) nutritious substance into tissues. It improves anabolic and catabolic processes which help absorption of energy. Yogic cleansing techniques promote elimination of ama (toxic products) and kleda (waste products). Yoga therapy along with herbal medicine and lifestyle modification helps develop balanced state of doshas in individuals with IBD. Yoga practice has a healing effect on mind and body, reduces stress, increases emotional and physical self-awareness, and improves the ability to manage physical symptoms.
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Pathogenesis Of Ibd

The imbalance of higher bodies, energy, mental, wisdom and bliss bodies alter biofeedback mechanism and the association of psychosocial factors, sociocultural factors, smoking, dietary habits and repeated use of drugs affects genetic predisposition which may lead to abnormal communication of gut-brain axis. Undseth et al. (2014) found that altered gut-brain activity resulted in significantly lower postprandial levels of total short chain fatty acids (SCFAs like Faecali bacterium prausnitzii and Roseburia intestinalis), acetic acid, propionic acid and butyric acid (Venegas et al., 2019). Bacteroidetes (gram-negative) mainly produce acetate and propionate while Firmicutes (gram-positive) mostly produce butyrate; both are abundant phyla in the human intestine. The main substrates for bacterial fermentation and SCFA production are resistant starch, inulin, pectin, cellulose, wheat bran, oat bran and guar gum from the non-digestible dietary fibres (NDDF). Anaerobic fermentation occurs in the NDDF produced which split into carbohydrate polymers with three or more monomeric units, that are neither digested nor absorbed in the human gut.

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