Welcome to the InfoSci Platform
Reference Hub1
ER Stress Signaling in Alzheimer's Disease: Molecular Mechanisms and Therapeutic Implications

ER Stress Signaling in Alzheimer's Disease: Molecular Mechanisms and Therapeutic Implications

Md. Motiar Rahman, Looniva Shrestha, Mst Ara Gulshan
Copyright: © 2020 |Pages: 32
ISBN13: 9781799813170|ISBN10: 1799813177|EISBN13: 9781799813187
DOI: 10.4018/978-1-7998-1317-0.ch007
Cite Chapter Cite Chapter

MLA

Rahman, Md. Motiar, et al. "ER Stress Signaling in Alzheimer's Disease: Molecular Mechanisms and Therapeutic Implications." Quality Control of Cellular Protein in Neurodegenerative Disorders, edited by Md. Sahab Uddin and Ghulam Md. Ashraf, IGI Global, 2020, pp. 180-211. https://doi.org/10.4018/978-1-7998-1317-0.ch007

APA

Rahman, M. M., Shrestha, L., & Gulshan, M. A. (2020). ER Stress Signaling in Alzheimer's Disease: Molecular Mechanisms and Therapeutic Implications. In M. Uddin & G. Ashraf (Eds.), Quality Control of Cellular Protein in Neurodegenerative Disorders (pp. 180-211). IGI Global. https://doi.org/10.4018/978-1-7998-1317-0.ch007

Chicago

Rahman, Md. Motiar, Looniva Shrestha, and Mst Ara Gulshan. "ER Stress Signaling in Alzheimer's Disease: Molecular Mechanisms and Therapeutic Implications." In Quality Control of Cellular Protein in Neurodegenerative Disorders, edited by Md. Sahab Uddin and Ghulam Md. Ashraf, 180-211. Hershey, PA: IGI Global, 2020. https://doi.org/10.4018/978-1-7998-1317-0.ch007

Export Reference

Mendeley
Favorite

Abstract

Alzheimer's disease (AD) is the most common etiology of dementia amongst aged individuals and a principal public health-related abnormality. It is considered as a multifactorial disorder, with no particular origin identified, and also some modifiable, as well as non-modifiable threats are correlated with its progression and development. The endoplasmic reticulum (ER) stress response is considered as a key process in the pathogenesis of AD. In this chapter, the authors present a summary of related transmembrane kinase proteins responsible for the onset of AD as well as show the interrelationship between ER stress and AD. Finally, the authors demonstrate the therapeutics intervention for AD diagnosis by highlighting the current practices to advance novel therapies.

Request Access

You do not own this content. Please login to recommend this title to your institution's librarian or purchase it from the IGI Global bookstore.